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Read More . . . TMG may be one of the most important nutrients to prevent heart disease, stroke, liver disease and to slow aging. TMG (trimethylglycine) may be one of the most important nutrients to prevent heart disease, stroke, liver disease and to slow aging. TMG is also called glycine betaine, but the name trimethylglycine signifies that it has three methyl groups attached to each molecule of glycine. In the body, TMG donates one of its methyl groups to cellular DNA, which may help protect DNA against damage and its consequences, cancer and premature aging. When a TMG methyl group is donated to a molecule of homocysteine, it is converted first to methionine then to S-adenosyl-methionine (SAMe). No safe level of homocysteine has been established, suggesting that taking nutrients like TMG could lower the risk of heart attack and stroke, in addition to protecting DNA. TMG stands for trimethylglycine which means it has three methyl groups attached to each molecule of glycine. In the body, TMG donates one of its methyl groups to cellular DNA, which may help protect DNA against pre-mature aging. When a TMG methyl group is donated to a molecule of toxic homocysteine, it converts the homocysteine to methionine and s- adenosyl-methionine. A safe level of homocysteine has not been established and this means that taking nutrients like TMG could lower everyone's risk of heart attack and stroke, in addition to protecting DNA. TMG should be taken with co-factors Vitamin-B12 and folic. If you take Life Extension Mix and Life Extension Booster, you will get these co- factors. Homocysteine, folate, and vascular disease Kannel W.B.; Wilson P.W.F. Framingham Heart Study, Boston University School of Medicine, Boston, MA USA Journal of Myocardial Ischemia (USA) , 1996, 8/2 (60-63) Current evidence indicates that the genesis of atherosclerotic disease is multifactorial. One of the newly recognized factors that contributes to this process is raised homocysteine blood levels. A variety of atherosclerotic processes may be facilitated by elevated homocysteine levels, including stimulation of smooth muscle cell growth, impairment of endothelial regeneration, oxidation of LDL particles, and thrombogenesis. A generic defect may account for some instances of hyperhomocysteinemia, but the majority of persons with high levels do not have known genetic defects to account for their elevations. Low levels of folic acid, vitamin B12, and pyridoxine appear to underlie most cases of elevated homocysteine levels. Adding folic acid to the diet may reduce homocysteine levels, but a link between increasing folic acid and lower risk of atherosclerotic disease has yet to be demonstrated in clinical trials. However, increasing daily folic acid intake is not unjustified in some patients. Since this may mask B12 deficiency, a supplement of cobalamin, 1 mg/d, has been proposed. In the final analysis, a clinical trial is needed to determine the true significance of hyperhomocysteinemia. Meanwhile, physicians and patients can consider increasing the daily folate intake by eating more oranges, leafy vegetables, wheat products, and cereals. How to Order To order online, click on the "Order" box below. To see a list of all supplements, please check our Index or use our Search feature. If you have any questions, please contact us at info@lifeextensionvitamins.com, 1-510-527-3005 or 1-888-771-3905. |
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